Aging
Verified
Journal
Aging publishes research papers in all fields of aging research including but not limited, organismal aging and cellular senescence, DNA damage repair, genetic control of aging from yeast to mammals, regulation of longevity, age-related diseases such as cancer and Alzheimer’s diseases and their prevention and treatment, anti-aging strategies and drug development and especially the role of signal transduction pathways such as mTOR in aging and potential approaches to modulate these signaling pathways to extend lifespan.
The journal aims to promote treatment of age-related diseases by slowing down aging, validation of anti-aging drugs by treating age-related diseases, prevention of cancer by inhibiting aging, and focusing on COVID-19 vulnerability as an age-dependent syndrome. Source
Actions
Media Outlet details
| Scope | National, Trade/B2B |
|---|---|
| Language | English |
| Country | United States of America |
Similarweb UVM
|
Request pricing |
Comscore UVM
|
Request pricing |
| Frequency | Biweekly/Fortnightly |
| Days Published | N/A |
| Accepts contributed content | Yes |
Recent Articles
Search ArticlesMultiple Biological Triggers Shape Cellular Senescence in Aging and Disease Original
“Cellular senescence is a stable form of cell-cycle arrest induced by diverse intrinsic and extrinsic stimuli.” BUFFALO, NY — July 7, 2026 — A new review was published in Volume 18 of Aging on June 22, 2026, titled “The multifaceted inducers of cellular senescence.” The review was led by first author Hilah Gal and corresponding author Valery Krizhanovsky from the Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel.
Comprehensive Individual-Patient Analysis Clarifies Life Expectancy Across Rare Progeria Disorders Original
“Classical laminopathic progeroid syndromes link nuclear-envelope defects to accelerated aging, but the specific prognosis for each subtype remains unclear.” BUFFALO, NY — July 6, 2026 — A new systematic review was published in Volume 18 of Aging on June 18, 2026, titled “Life expectancy and causes of death in classical laminopathic progeroid syndromes: systematic review with individual-patient data synthesis.” The study was led by co-first authors Carlos López-Vila, Manuel García-Cordeiro,...
Peer-Reviewed Aging Research Journal | Aging
Rapamycin Press LLC dba Impact Journals is the publisher of Aging: www.impactjournals.com. Impact Journals is a member of the Wellcome Trust List of Compliant Publishers. Impact Journals is a member of the Society for Scholarly Publishing.
Inhibition of peroxisomal protein PRX-11 promotes longevity in Caenorhabditis elegans via enhancements to mitochondria
Research Paper|Volume 18|pp 787—812 1Department of Biological Sciences, Louisiana State University, Baton Rouge, LA 70803, USA Received: May 30, 2025Accepted: May 13, 2026Published: July 6, 2026 Copyright: © 2026 Flora et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Nonhuman Primate Research Reveals How Aging, Stress, and Behavior May Interact to Increase Disease Risk
“With age, the incidence of stress-related pathologies, in the etiopathogenesis of which endocrine dysfunctions play an important role, increases.” BUFFALO, NY — July 1, 2026 — A new review was published in Volume 18 of Aging on June 11, 2026, titled “Age-related dysfunctions of the neuroendocrine axes in nonhuman primates with depression-like and anxious behavior.” The review, dedicated to the late Dr. Mikhail (Misha) Blagosklonny, was written by Nadezhda D.
Senescent cells accumulate lipid droplets
Introduction Cellular senescence is a state of cell cycle arrest in previously proliferating cells, triggered by cellular stress [1–3]. This complex biological process can exert both beneficial and detrimental effects in a biological context-dependent manner [1–3]. Cellular senescence acts as a barrier to tumorigenesis by limiting the excessive proliferation of damaged cells [4].
Cellular Senescence and Stem Cells Share a More Complex Relationship Than Previously Recognized
“Cellular senescence and stemness represent two biological entities with several opposing properties.” BUFFALO, NY — June 29, 2026 — A new review was published in Volume 18 of Aging on May 30, 2026, titled “The love and hate relationship between cellular senescence and stemness.” The review was led by first author Angelos Papaspyropoulos and corresponding author Vassilis G.
Hormonal dimorphism in sarcopenia disease
Review|Volume 18|pp 733—767 1MDI Biological Laboratory, Kathryn W. Davis Center for Regenerative Biology and Aging, Bar Harbor, Maine 04609, USA Received: February 9, 2026Accepted: May 13, 2026Published: June 26, 2026 Copyright: © 2026 Menard and Madelaine.
Lifestyle Intervention Linked to Slower Biological Aging Markers in Older Men Original
“The intervention group exhibited a significant deceleration in DunedinPACE, corresponding to an estimated 2.2% slower pace of aging, whereas no meaningful change was observed in the control group.” BUFFALO, NY — June 25, 2026 — A new research paper was published in Volume 18 of Aging on May 29, 2026, titled “Short-term responsiveness of DNA methylation–based aging biomarkers to a multimodal intervention comprising exercise and dietary guidance involving daily consumption of yogurt containing...
Researchers Identify Key Signaling Pathway Driving Muscle Cell Aging
“Skeletal muscle displays an age-associated decline in motor function and muscle mass.” BUFFALO, NY — June 23, 2026 — A new research paper was published in Volume 18 of Aging on May 28, 2026, titled “p38MAP kinase regulates senescence in human iPS-derived myocytes.” The study was led by first author Hiroki Sato and corresponding author Ryuichiro Sato from The University of Tokyo, along with corresponding author Makoto Shimizu from The University of Tokyo and Ochanomizu University.