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Articles by A. Ravandi
RELATIONSHIP BETWEEN PARAMETERS DEFINING CARDIOGENIC SHOCK AND OUTCOME IN PATIENTS WITH ST-ELEVATION MYOCARDIAL INFARCTION
Despite advances in care, cardiogenic shock (CS) remains the leading cause of death in patients with ST-elevation myocardial infarction (STEMI). One plausible reason we have failed in treating this high-risk cohort effectively may lie in our ability to objectively define CS and intervene timely. Per current guidelines, CS is defined as either (1) systolic blood pressure (SBP) ≤ 90mmHg, (2) cardiac-index (CI) < 1.8 L/min/m2 or 2 mmol/L; these parameters are used interchangeably.
SEX DIFFERENCES IN PLASMA LIPIDOME IN PATIENTS WITH STEMI UNDERGOING PCI
Rapid coronary reperfusion using primary percutaneous coronary intervention (PCI) is the corner stone of therapy for patients presenting with ST elevation myocardial infarction (STEMI) that restores blood flow and limits infarct size. Paradoxically, however, PCI in itself can induce additional myocardial injury, termed reperfusion injury. We have recently shown that there are large shifts in plasma lipidome post PCI correlating with clinical outcomes.
QUANTITATIVE PROFILING OF OXYLIPINS IN PATIENTS PRESENTING WITH ST ELEVATION MYOCARDIAL INFARCTION (STEMI)
To view the full text, please login as a subscribed user or purchase a subscription. Click here to view the full text on ScienceDirect. Previous studies have shown that there is persistent oxidative stress after myocardial infarction (MI) and percutaneous coronary intervention (PCI). Oxylipins are lipid oxidation products that can be generated mainly by enzymatic oxidation through cyclooxygenase (COX), lipoxygenase (LOX) and cytochrome P450 (CYP P450) pathways.
TARGETED LIPIDOMIC ANALYSIS OF KIDNEY DURING ISCHEMIA AND REPERFUSION INJURY: IDENTIFICATION OF BIOACTIVE LIPIDS
To view the full text, please login as a subscribed user or purchase a subscription. Click here to view the full text on ScienceDirect. Ischemia/reperfusion (I/R) leads to series of cellular responses such as release of reactive oxygen species, elevation of apoptotic molecules, necrosis, inflammation and release of bioactive compounds cause structural damage. Oxidized phosphatidylcholines (OxPCs) are the most abundant compounds produced during reperfusion due to oxidative stress.
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By Shari Liu Verified, B. Hiebert, N. Tangri, A. Ravandi, O. Toleva, B. Elbarouni, M. Vo, K. Minhas, J. Ducas, C. Rigatto, M. Kass
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