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1 Introduction The role of subgingival bacterial dysbiosis in the initiation and progression of periodontal diseases is well documented (Hajishengallis et al. 2012, 2015, 2020). Dysbiotic bacterial alterations that involve increased levels of Porphyromonas gingivalis, Treponema denticola, and Tannerella forsythia in subgingival plaque have been associated with increased risk for destructive periodontitis (Socransky et al. 1998; Darveau 2010; Suzuki et al. 2013).
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2.6 Statistical analysis This is a convenience sample of a previously described periodontitis-resistant cohort (Khocht et al., 2021). Twenty subjects were lean (BMI < 25), 11 subjects were overweight (BMI between 25 and 30) and 8 subjects were obese (BMI ≥ 30). For data analysis purposes, overweight and obese subjects were combined into a single group. The distributions of clinical periodontal measures, metabolite concentrations and relative abundance of microbial taxa were positively skewed.
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