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1 Introduction Chronic hepatitis B virus (HBV) infection is a major cause of liver cirrhosis and hepatocellular carcinoma (HCC) [1, 2]. The course of the infection is highly variable, and the main task for clinicians is to identify patients who are at risk for complications, in order to prevent necroinflammation and HCC by antiviral therapy and identify curable HCC by screening programs.
The level of hepatitis B virus RNA in the blood has become established as a clinical marker. In this study, we found by quantifying both DNA and RNA in three parts of the HBV genome that particles with HBV RNA are not fundamentally different from those with HBV DNA but represent viruses with incomplete reverse transcription.
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