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Abstract Previous research suggests that older adults may display more prosocial behavior than younger adults. However, recent meta-analyses indicate that effects are heterogeneous, may be small, and are influenced by how prosociality is measured. Further, the precise cognitive and computational factors contributing to age-related differences in prosocial behavior remain largely unknown.
Abstract Cognition and brain structure undergo significant maturation from adolescence into adulthood. Model-based (MB) control is known to increase across development, which is mediated by cognitive abilities. Here, we asked two questions unaddressed in previous developmental studies. First, what are the brain structural correlates of age-related increases in MB control? Second, how are age-related increases in MB control from adolescence to adulthood influenced by motivational context?
Abstract Cognition and brain structure undergo significant maturation from adolescence into adulthood. Model-based (MB) control is known to increase across development, which is mediated by cognitive abilities. Here, we asked two questions unaddressed in previous developmental studies: Firstly, what are the brain structural correlates of age-related increases in MB control? Secondly, how are age-related increases in MB control from adolescence to adulthood influenced by motivational context?
Introduction Infection with high-risk human papillomavirus (HPV) types is a major cause of head and neck squamous cell carcinoma (HNSCC), cervical and anogenital cancers. HPV16, the most prevalent HPV type, accounts for the majority of these cancers, 1,2 in which surgery, chemotherapy and radiotherapy are the traditional standard of care.
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