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Articles by Andreas Daiber
Reply to ‘Distinguishing between nicotine replacement therapies and nicotine-containing consumer products’
We thank Berlin and Thomas for their Correspondence on our Review (Münzel, T. et al. Nicotine-containing products and the cardiovascular system: mechanisms and global health implications. Nat. Rev. Cardiol. https://doi.org/10.1038/s41569-026-01331-6; 2026)1 and for the issues raised (Berlin, I. & Thomas, D. Distinguishing between nicotine replacement therapies and nicotine-containing consumer products. Nat. Rev. Cardiol.
Nicotine-containing products and the cardiovascular system: mechanisms and global health implications
Abstract Tobacco use and emerging nicotine-delivery systems remain major, preventable drivers of cardiovascular disease worldwide. Although tobacco cigarette smoking has declined in some high-income regions, progress is uneven, with persistently high rates across parts of Europe, alongside rapidly expanding global markets for e-cigarettes, heated-tobacco products and oral nicotine formulations.
Nikotin schädigt das Herz – die Politik schaut zu
Nikotin ist ein eigenständiger und häufig unterschätzter kardiovaskulärer Risikofaktor. Der überwiegende Teil der nikotinassoziierten Mortalität ist Herz-Kreislauf-bedingt und tritt oft früher auf als Krebsfolgen. Ein konsistentes Frühwarnsignal ist die endotheliale Dysfunktion – ein valider Prädiktor künftiger Ereignisse, der nur bei vollständiger Nikotin-Abstinenz reversibel ist. Trotz klarer Evidenz bleibt heute die Regulierung fragmentiert und politisch unzureichend.
Absence of MCJ/DnaJC15 promotes brown adipose tissue thermogenesis - Nature Communications
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Abstract Obesity poses a global health challenge, demanding a deeper understanding of adipose tissue (AT) and its mitochondria. This study describes the role of the mitochondrial protein Methylation-controlled J protein (MCJ/DnaJC15) in orchestrating brown adipose tissue (BAT) thermogenesis. Here we show how MCJ expression decreases during obesity, as evident in human and mouse adipose tissue samples.
By Beatriz Cicuéndez, Juan Lopez, Juan López, Andrea Curtabbi, Javier Perez-Garcia, Daniel Jimenez-Blasco, Paula Vo, Beatriz Gomez-Santos, Rafael Romero-Becerra, Jorge Torres, Miguel Marcos Verified, Martin Jastroch, Andreas Daiber, Patricia Aspichueta, Juan Pedro Bolaños, Jessica B. Spinelli, Pere Puigserver, José Antonio Enríquez, Jesús Vázquez Verified, Cintia Folgueira, Lourdes Hernández-Cosido
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Nature
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Deutsche Medizinische Wochenschrift / Abstract
Die zunehmende Häufigkeit und Intensität von Waldbränden, verstärkt durch den Klimawandel, stellt eine beträchtliche Bedrohung für Umwelt und Gesundheit dar. Diese Brände verursachen eine erhebliche Luftverschmutzung – insbesondere durch Feinstaubpartikel. Dieser Beitrag beleuchtet, auf welche Weise Waldbrandrauch die Gesundheit gefährdet, welche Bevölkerungsgruppen davon besonders betroffen sind und welche Maßnahmen ergriffen werden können.
Crosstalk between Oxidative Stress and Inflammation Caused by Noise and Air Pollution-Implications for Neurodegenerative Diseases
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Endogenous Nitric Oxide Can Enhance Oxidative Stress Caused by Air Pollutants and Explain Higher Susceptibility of Individuals with Inflammatory Disorders
Download Hi-Res ImageDownload to MS-PowerPointCite This:Environ. Sci. Technol.
Endogenous Nitric Oxide Can Enhance Oxidative Stress Caused by Air Pollutants and Explain Higher Susceptibility of Individuals with Inflammatory Disorders
Download Hi-Res ImageDownload to MS-PowerPointCite This:Environ. Sci. Technol.
Noise, Air, and Heavy Metal Pollution as Risk Factors for Endothelial Dysfunction
Endothelial Dysfunction Is Induced by Environmental Stress The inability of a vessel to dilate fully, which arises from a decrease in NO bioavailability caused by reduced production, scavenging by reactive oxygen species (ROS), or both, is known as endothelial dysfunction.24 During the last decades, it has been shown that novel risk factors in the physical environment may facilitate the development of cardiovascular complications such as endothelial dysfunction.31,32 For this reason,...
Physical activity in polluted air: an urgent call to study the health risks
Both physical inactivity and exposure to air pollution rank among the most substantial public health threats, contributing to increased premature morbidity and mortality due to non-communicable diseases. Globally, it is estimated that one in four adults is physically inactive, meaning that the minimum recommended amounts of physical activity are not met (150 min of moderate-intensity physical activity or 75 min of vigorous-intensity physical activity per week).
Running in Polluted Air Is a Two-edged Sword
Authors and Disclosures Thomas Münzel1,2*, Omar Hahad1,2 and Andreas Daiber1,2 1Department of Cardiology, Cardiology I, University Medical Center Mainz, Mainz, Germany; and 2German Center for Cardiovascular Research (DZHK), Partner Site Rhine-Main, Mainz, Germany *Corresponding author University Medical Center Mainz, Department of Cardiology, Cardiology I, Geb. 605, Langenbeckstr. 1, D-55131 Mainz, Germany.
Transportation noise pollution and cardiovascular disease - Nature Reviews Cardiology
Abstract Epidemiological studies have found that transportation noise increases the risk of cardiovascular morbidity and mortality, with high-quality evidence for ischaemic heart disease. According to the WHO, ≥1.6 million healthy life-years are lost annually from traffic-related noise in Western Europe. Traffic noise at night causes fragmentation and shortening of sleep, elevation of stress hormone levels, and increased oxidative stress in the vasculature and the brain.
Could E-cigarette vaping contribute to heart disease?
Invalidity of an oft-cited estimate of the relative harms of electronic cigarettes • • Important reading providing a rationale for the invalid “95% less harmful” assertion concerning E-cigarettes.
Could E-cigarette vaping contribute to heart disease?
ABSTRACT Introduction E-cigarettes have become a controversial topic. While their benefits are questioned by the scientific community, a part of the medical profession is still supporting them as an effective harm reduction tool for smoking cessation. The impact of E-cigarettes on the cardiovascular system is still elusive.
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