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We first assessed the expression of TWEAK and Fn14 in the skin in experimental AD. This was induced by repetitive topical exposure to house dust mite (HDM) allergen and staphylococcal enterotoxin B (SEB)26,27, both implicated in the pathogenesis of AD in humans28,29,30. Dermatitis develops in a T-cell-dependent manner31 with involvement of type 2 cytokines, including TSLP32, that are characteristic of human AD.
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