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Hypothermia prevented secondary drop in mCBC and attenuated HI brain injury Immediately after HI and prior to the reperfusion, mitochondrial Ca2+ retention capacity at 25°C was significantly decreased compared to controls (Fig 2A and 2B). At 30 minutes of normothermic or hypothermic reperfusion, in NT and HT mice, their mCBCs have significantly recovered and did not differ from that in controls (Fig 2A and 2C).
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