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Abstract Oncolytic viruses (OVs) preferentially infect and replicate within tumors, induce immunogenic cell death, and remodel immune-poor microenvironments to support T-cell infiltration. These features provide a rationale for combining OVs with immune checkpoint blockade, adoptive cellular therapies, bispecific T-cell engagers, cancer vaccines, and innate immune agonists, and for engineering viruses to express immunostimulatory cytokines. Clinical outcomes, however, have been inconsistent.
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