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Abstract Background: Vision loss in bacterial keratitis arises primarily from the host inflammatory response rather than the infection itself, and antimicrobial resistance narrows the benefit achievable with antibiotics alone. AZD6738, an inhibitor of ataxia telangiectasia and Rad3-related kinase (ATR), suppresses lipopolysaccharide (LPS)- induced corneal inflammation in mice, but the transcriptional basis of this effect is uncharacterized.
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