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Articles by Brinkley A. Morse

Virus reactivation in acute and long COVID-19

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By Cole Maguire, JING CHEN, Nadine Rouphael, Annmarie Hoch, Harry Pickering, Hoang Van Phan, Abigail Glascock, Victoria M. Chu, Ravi Dandekar, David Corry, Farrah Kheradmand, Lindsey R. Baden, Rafick-Pierre Sekaly, Grace A McComsey, Elias Haddad, Charles B. Cairns, Ana Fernández-Sesma, Viviana Simon, Jordan P Metcalf, William Messer, Mark Davis Verified, Kari C. Nadeau, Monica Kraft, Chris Bime, Joanna Schaenman, David J. Erle, Carolyn S Calfee, Mark Atkinson Verified, Scott C Brakenridge, Lauren I. R. Ehrlich, Ruth R. Montgomery, Albert Shaw, Catherine L. Hough, David A. Hafler, Alison D. Augustine, Patrice Becker, Bjoern Peters, Al Ozonoff, Seunghee Kim-schulze, Florian Krammer, Steven E. Bosinger, Walter L Eckalbar, Matthew Altman, Michael Wilson Verified, Steven Kleinstein, Kinga K. Smolen, Elaine Reed, Ofer Levy, Holden T Maecker, Peter Hunt, Hanno Steen, Joann Diray-Arce, Charles Langelier, Esther Melamed, Brinkley A. Morse, Nelson I. Agudelo Higuita
| Nature Verified
Abstract Chronic viral infections are ubiquitous in humans, with individuals carrying multiple viruses that can reactivate during physiological stress, including severe illness1. Notably, SARS-CoV-2 infection has been shown to reactivate chronic viruses such as Epstein–Barr virus and cytomegalovirus, yet the full extent, temporal dynamics and immunological impact of viral reactivation in COVID-19 remain incompletely understood2,3,4,5,6,7.
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