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Abstract Metabolic dysfunction-associated steatotic liver disease (MASLD) is the most prevalent chronic liver disease worldwide, yet no standardized pharmacotherapy has been approved for clinical use. The autophagy–lysosome pathway (ALP) serves as an essential intracellular degradation and recycling machinery that sustains hepatic metabolic homeostasis via modulation of lipid turnover, mitochondrial quality surveillance, and hepatocellular detoxification.
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