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Abstract Sodium-glucose cotransporter 2 (SGLT2) inhibitors provide renal protection in diabetic kidney disease (DKD) through mechanisms beyond glucose lowering, but these remain incompletely understood. Here we show that Yes-associated protein 1 (YAP)-dependent ferroptosis contributes to renal tubular injury in DKD and that suppression of this pathway underlies part of the renoprotective effects of dapagliflozin.
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