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Abstract Introduction: Resistance to tyrosine kinase inhibitors (TKIs) remains a major cause of treatment failure in chronic myeloid leukemia (CML). This study investigated the role of ribosomal protein S3 (RPS3) in TKI resistance and its association with NF-κB signaling. Methods: Exploratory RNA sequencing and an RNA-binding protein database were used to prioritize a candidate for subsequent clinical expression and functional analyses.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
