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1 Introduction Cellular senescence is marked by the inability of normal cells to divide, resulting in a state of irreversible cell cycle arrest. As senescence progresses, non-dividing cells secrete senescence-associated secretory phenotype (SASP) factors, which promote the spread and accumulation of senescent cells in tissues [1, 2]. Normal cells self-destruct through apoptosis, which is difficult to repair due to genetic damage.
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