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Daniel Eitzman

As seen in: Nature, PLOS ONE
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Articles by Daniel Eitzman

Empagliflozin attenuates traumatic brain injury-induced atherosclerosis and norepinephrine-induced vascular smooth muscle cell responses

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By Jintao Wang, Enming J. Su, Daniel Lawrence, Daniel Eitzman
| Nature Verified
Abstract Traumatic brain injury (TBI) is associated with increased long-term cardiovascular risk, but strategies to limit post-traumatic vascular disease are lacking. We tested whether empagliflozin, a sodium-glucose cotransporter 2 inhibitor, attenuates TBI-induced atherosclerosis in apolipoprotein E-deficient mice. Mice on a Western diet underwent controlled cortical impact or sham surgery and received empagliflozin or vehicle for 10 weeks.
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Resolution of Sickle Cell Crisis Following Administration of Amiodarone

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By Eitzman DT, Paul Silaghi, Daniel Eitzman
| ncbi.nlm.nih.gov
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Revascularization in Patients With Spontaneous Coronary Artery Dissection: Where Are We Now?

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By Chayakrit Krittanawong, Rajiv Gulati, Hani Jneid, Daniel Eitzman
| AHA/ASA Journals
Spontaneous coronary artery dissection (SCAD) is a heterogeneous condition that often presents as an acute coronary syndrome in young patients with a paucity of cardiovascular risk factors. In most registries, >90% of patients with SCAD are women.1 SCAD is primarily a non‐atherosclerotic, non‐calcified, non‐iatrogenic dissection of the coronary artery resulting from either an intramural hematoma (IMH) alone, an intimal tear alone, or both an IMH and an intimal tear.
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