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1 Introduction Sickle cell disease (SCD) affects approximately 100 000 people in the U.S. Caused by a single amino acid substitution in the β hemoglobin gene, normal hemoglobin A is changed to hemoglobin S (HbS). HbS polymerization leads to sickled, rigid red blood cells (RBC) which produce chronic hemolysis and end organ damage [1].
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