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Abstract Silk fibroin (SF) hydrogels have emerged as versatile matrices for tissue repair, supporting injectable delivery, biofabrication and implantation while offering tunable mechanics, mass transport and biodegradation. Repair outcomes, however, are often dictated by the local microenvironment, where prolonged inflammation, oxidative stress, infection, impaired vascularization, and mechanical mismatch can drive fibrosis and limit functional integration.
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