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Articles by Dmitry Troshev
Changes in gene expression of tyrosine hydroxylase, its regulating transcription factors, dopamine receptors in striatal neurons after their dopaminergic denervation
Abstract Among the priorities in neurology is the development of pathogenetic treatment for Parkinson’s disease (PD). Striatal neurons capable of expressing the tyrosine hydroxylase gene (Th) and containing this enzyme represent a potential target for such therapy. Transgenic mice with the green fluorescent protein gene under the Th promoter were used to model PD via 1‑methyl‑4‑phenyl‑1,2,3,6‑tetrahydropyridine neurotoxin.
Salt loading as a promising approach to study the dopaminergic phenotype of neurons of the supraoptic nucleus in mice
Loading metrics Open Access Peer-reviewed Research Article Citation: Bannikova AE, Pronina TS, Troshev DV, Bogdanov VV, Kolacheva AA, Pavlova EN, et al. (2026) Salt loading as a promising approach to study the dopaminergic phenotype of neurons of the supraoptic nucleus in mice. PLoS One 21(2): e0340281.
Genes | Free Full-Text | Application of OpenArray Technology to Assess Changes in the Expression of Functionally Significant Genes in the Substantia Nigra of Mice in a Model of Parkinson’s Disease
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Compensatory Processes in Striatal Neurons Expressing the Tyrosine Hydroxylase Gene in Transgenic Mice in a Model of Parkinson’s Disease
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Neuroprotective Efficacy of a Nanomicellar Complex of Carnosine and Lipoic Acid in a Rat Model of Rotenone-Induced Parkinson’s Disease
Abstract : Oxidative stress, accompanied by mitochondrial dysfunction, is a key mechanism involved in the pathogenesis of Parkinson’s disease (PD). Both carnosine and lipoic acid are potent antioxidants, the applicability of which in therapy is hindered by their limited bioavailability. This study aimed to evaluate the neuroprotective properties of a nanomicellar complex of carnosine and lipoic acid (CLA) in a rotenone-induced rat model of PD.
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