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Abstract Inflammatory disease is sustained not only by immune activation but also by the failure of activated cells to terminate effector programs and return tissues to homeostasis. This review evaluates N6-methyladenosine (m6A) as a post-transcriptional regulator of that persistence.
Abstract YTH N6-methyladenosine RNA binding protein F1 (YTHDF1) enables N6-methyladenosine-containing RNA binding activity, involved in mRNA destabilization and positive regulation of translational initiation. Here we aimed to investigate the main molecular events associated with YTHDF1 during the course of metabolic dysfunction-associated steatotic liver disease (MASLD). YTHDF1 expression was detected by western blotting and real-time PCR.
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