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Articles by Einar Eftestøl
Mechanisms of Skeletal Muscle Mass Regulation and Muscle Memory
Conflicts of Interest The authors declare no conflicts of interest. Data Availability Statement The data that support the findings of this study are available from the corresponding author upon reasonable request. References 1, , , and , “Muscle Fiber Splitting Is a Physiological Response to Extreme Loading in Animals,” Exercise and Sport Sciences Reviews 47, no. 2 (2019): 108–115. 2, “Effects of Aging on Muscle Fibre Type and Size,” Sports Medicine 34, no. 12 (2004): 809–824.
No detectable loss of myonuclei from human muscle fibers after 6 wk of immobilization following an Achilles tendon rupture
Skeletal muscle is a malleable tissue with a capacity to alter its phenotype in response to different types of loading. One well-known negative regulator of muscle mass is the absence of mechanical loading, i.e., disuse, which leads to the deterioration of muscle mass and function (1). This process naturally occurs in the context of musculoskeletal injuries to protect the injured limb from further injury and to allow for healing.
A cellular memory mechanism aids overload hypertrophy in muscle long after an episodic exposure to anabolic steroids
K. Gundersen: Department of Biosciences, University of Oslo, PO Box 1066 Blindern, N‐0316 Oslo, Norway. Email: kgunder@ibv.uio.no I. M. Egner and J. C. Bruusgaard contributed equally to this work. PDF ShareShare Key points • Training studio folklore suggests that previous strength training, with or without the use of anabolic steroids facilitates re‐acquisition of muscle mass even after long intervening periods of inactivity.
EFFECTS OF TRAINING, DETRAINING, AND RETRAINING ON STRENGTH, HYPERTROPHY, AND MYONUCLEAR NUMBER IN HUMAN SKELETAL MUSCLE
Research Article Niklas Psilander, Einar Eftestøl, Kristoffer Toldnes Cumming, Inga Juvkam, Maria M. Ekblom, Kerstin Sunding, Mathias Wernbom, Hans-Christer Holmberg, Bjorn Ekblom, Jo C. Bruusgaard, Truls Raastad, and Kristian Gundersen Abstract Previously trained mouse muscles acquire strength and volume faster than naïve muscles; it has been suggested that this is related to increased myonuclear density.
A cellular memory mechanism aids overload hypertrophy in muscle long after an episodic exposure to anabolic steroids
Key points • Training studio folklore suggests that previous strength training, with or without the use of anabolic steroids facilitates re‐acquisition of muscle mass even after long intervening periods of inactivity. This ‘muscle memory’ has previously been attributed to motor learning, but our data suggest the existence of a cellular memory residing in the muscle fibres themselves. • Muscle fibres have multiple nuclei, and the number of nuclei increases when muscle mass increases.
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