INTRODUCTION Swallowing is a complex task, moderated by a sophisticated bilateral network including the cerebral cortices, subcortical regions, brainstem and cerebellum. Hence, lesions to this widely distributed swallowing network might be expected to result in acute post-stroke dysphagia. In this context, numerous studies showed that the incidence, severity and recovery of dysphagia after stroke is related to lesion location and size. Moreover, a recent study by Warnecke et al.
