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1 Introduction Anti-N-methyl-D-aspartate receptor encephalitis (NMDAR-E) is among the most prevalent and clinically consequential autoimmune neurological disorders. Over the past two decades, understanding of NMDAR-E has evolved substantially, and the disease is now recognized as a well-defined clinical entity [1, 2]. Substantial progress has concurrently been achieved in targeted immunotherapeutic interventions and symptom-directed management approaches [3, 4].
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