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Abstract Allergic rhinitis (AR) has traditionally been regarded as a type I hypersensitivity disorder confined to the nasal mucosa. However, persistent symptoms, nasal hyperresponsiveness, and comorbid neuropsychiatric disturbances—including headache, fatigue, cognitive impairment (“brain fog”), anxiety, and depression—cannot be fully explained by peripheral inflammation alone, thereby implicating the involvement of central sensitization.
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