1 INTRODUCTION Idiopathic membranous nephropathy (IMN) is a common pathological type of glomerular disease, ranking second among primary glomerular diseases, with an increasing incidence rate. The histological hallmark of IMN is the deposition of immune complexes in the subepithelial space of the glomerular filtration barrier. Untreated IMN patients have a 10-year renal survival rate of 60%–80%.1 Over the past decade, there has been significant progress in understanding the pathogenesis of IMN.
