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1 Introduction Hidradenitis suppurativa (HS) is a chronic inflammatory dermatosis with over half of patients reporting symptom onset during adolescence [1, 2]. Despite this, HS treatment guidelines have only recently begun to emerge and management remains challenging [3]. Effective pediatric HS care should address comorbidities and physiologic differences [4].
Hidradenitis suppurativa (HS) is a chronic inflammatory disease. Numerous studies have associated HS with obesity, and recently with metabolic syndrome (MetS). Both obesity and MetS are linked with metabolic changes. Thyroid hormones play a central role in metabolism and exert pleiotropic effects on adipogenesis and the basal metabolism of lipids and glucose. We hypothesized that patients with HS have an altered or dysfunctional metabolism expressed as thyroid function.
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