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Abstract Neuroinflammation is an active and heterogeneous component of Alzheimer’s disease, shaped by dynamic interactions among microglia, astrocytes, neurons, and brain-interface compartments. Noncoding RNAs, including microRNAs, long noncoding RNAs, circular RNAs, and emerging small-RNA species, regulate innate immune sensing, interferon responses, phagocytosis, metabolic adaptation, inflammasome activity, and neuronal stress.
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