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Abstract Innate immunity significantly contributes to biliary atresia (BA) pathogenesis. Toll-like receptor 8 (TLR8) is an important innate immune mediator that is implicated in liver fibrosis. This study aimed to investigate the underlying mechanistic role of TLR8 in hepatic inflammation and fibrosis in BA. Transcriptomic analysis was performed on liver tissues from 21 BA patients and 18 non-tumor liver tissues taken from hepatoblastoma patients.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
