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Abstract E1A-binding protein p300 (p300)/CREB-binding protein (CBP) and phosphorylated Smad2 (pSmad2C/L) are highly activated in hepatocellular carcinoma (HCC), but their relationship remains unclear. Salvianolic (Sal B) exhibits potential anti-HCC activities. We previously reported Sal B downregulates p300/CBP and pSmad2C/L, thereby inhibiting hepatic stellate cell activation and exerting anti-fibrotic effects.
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