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Abstract The bispecific antibody IMV-M was designed to bind and cluster Death Receptor 5 (DR5) specifically upon engaging the tumor antigen MUC16. This dual-binding mechanism induces apoptosis in MUC16-positive tumor cells through a novel enhancement process. IMV-M exhibited potent, MUC16-selective anti-tumor activity in vitro and in xenograft models, achieving efficacy at safe therapeutic doses without requiring secondary crosslinking.
This article has been expanded and adapted from prepared remarks delivered at the Researchers.One panel “The Science and Social Impact of COVID-19 Vaccines” on March 25th, 2021. The current state of COVID-19 vaccine administration in the United States, with its high levels of vaccine hesitancy and skepticism, can be explained by a model composed of three layers of interconnected system(s) malfunction.
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