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Jan Piet

As seen in: Allergy - European Journal of Allergy and Clinical Immunology, Color Research & Application, Arthritis Research & Therapy, Trouw
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Articles by Jan Piet

Modulating IL-21-driven B cell responses in idiopathic inflammatory myopathies via inhibition of the JAK/STAT pathway - Arthritis Research & Therapy

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By van Leeuwen, Van der Kooi, Jan Piet, van Hamburg
| Arthritis Research & Therapy Verified
References Lundberg IE, Fujimoto M, Vencovsky J, Aggarwal R, Holmqvist M, Christopher-Stine L, et al. Idiopathic inflammatory myopathies. Nat Rev Dis Primers. 2021;7(1):86. PubMed Google Scholar Connolly CM, Gupta L, Fujimoto M, Machado PM, Paik JJ. Idiopathic inflammatory myopathies: current insights and future frontiers. Lancet Rheumatol. 2024;6(2):e115–27. CAS PubMed Google Scholar Schmidt J. Current classification and management of inflammatory myopathies. J Neuromuscul Dis. 2018;5(2):109–29.
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Maak van regio Schiphol een nieuw groen hart

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By Jan Piet
| Trouw Verified
Hoe zou je de eeuwige discussie over de groei van Schiphol in betere banen kunnen leiden? Want opnieuw, voor de zoveelste keer, gaan politici en bestuurders een bedrijfsmatig gezonde ontwikkeling van de luchthaven sturen door grenzen te stellen aan wat bewoners aan overlast ervaren. Dat is mijns inziens een doodlopende weg. Veel beter is het om in te zetten op leefklimaat. Vorige maand besloot het kabinet dat Schiphol onder voorwaarden mag groeien tot 540.000 vliegbewegingen per jaar.
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Loss of IL-22 inhibits autoantibody formation in collagen-induced arthritis in mice

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By Odilia B. J, Adriana M. C., Jan Piet
| Allergy - European Journal of Allergy and Clinical Immunology Verified
Interleukin 22 (IL-22) expression is associated with increased joint destruction and disease progression in rheumatoid arthritis (RA). Although IL-22 is considered a pro-inflammatory cytokine, its mechanism of action in RA remains incompletely understood. Here, we used the collagen-induced arthritis model in IL-22 deficient (IL-22−/−) mice to study the role of IL-22 in RA. In spite of normal disease incidence, disease severity is significantly diminished in IL-22−/- mice.
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