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Abstract Exercise intolerance in heart failure (HF) reflects combined cardiac and skeletal muscle abnormalities, but the molecular associations underlying concurrent remodeling remain unclear. We examined cardiac and skeletal muscle phenotypes in a rat model of ischemic HF with global miR-133a deficiency and explored candidate downstream signaling.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
