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The EA mechanism in promoting angiogenesis in CIRI rats Our analysis showed that EA at GV26 acupoint mitigated the ischemia/reperfusion injury of MCAO rats through promotion of angiogenesis in brain tissues. Angiogenesis did not only save the damaged neurons in the penumbra by increasing collateral circulation, but also provided the necessary material basis for neurogenesis and synapse formation which accelerated the recovery of neural function.
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