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Abstract Nuclear homeostasis requires a balance of forces between the cytoskeleton and nucleus. Variants in LMNA disrupt this balance by weakening the nuclear lamina, resulting in nuclear damage in contractile tissues and ultimately muscle disease. Intriguingly, disrupting the LINC complex that connects the cytoskeleton to the nucleus has emerged as a promising strategy to ameliorate LMNA cardiomyopathy. Yet how LINC disruption protects the cardiomyocyte nucleus remains unclear.
As a scientist, I understand how important it is that our work is shared with the public (it’s how we get our funding, after all). But the line of communication becomes muddled when journalists – despite their best intentions – misinterpret and oversimplify scientific studies and articles, leading to published articles that are often full of misinformation or oversimplifications. One doesn’t even need to fully read the article to be influenced.
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