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This is an unedited manuscript that has been accepted for publication. NPG are providing this early version of the manuscript as a service to our customers. The manuscript will undergo copyediting, typesetting and a proof review before it is published in its final form. Please note that during the production process errors may be discovered which could affect the content, and all legal disclaimers apply. PI3K?
On the basis of the key role of PI3K? in these processes, we hypothesized that PI3K?i disrupt multiple pro-survival inputs culminating in Bim-mediated intrinsic apoptosis and in vivo clearance of malignant cells. Although prior studies have been performed assessing PI3K?i-mediated immunomodulation, only limited data are available assessing the impact of PI3K? inhibition within a malignant target in vivo .
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