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Our previous studies have shown that in mice induced with EAE, the absence of Bcl11b in mature T-cells caused increased plasticity of Th17 cells by acquisition of GATA3 and IL-430. However, we found that Bcl11b plays a critical role in ILC2s, promoting their program and preserving their identity33. Given these results, we investigated the role of Bcl11b during a house dust mite (HDM)-induced asthma, a Th2-mediated disease.
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