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Breast cancer is a disease marked by cellular diversity, and cancer cells from the same primary tumor can colonize multiple metastatic sites. Metastasis is a rare and stochastic process, and because of that it becomes very difficult to predict when, where, and whether a patient diagnosed with breast cancer will develop metastases and which organs will be affected (1).
Understanding the rewired metabolism underlying organ-specific metastasis in breast cancer could help identify strategies to improve the treatment and prevention of metastatic disease. Here, we used a systems biology approach to compare metabolic fluxes used by parental breast cancer cells and their brain- and lung-homing derivatives. Divergent lineages had distinct, heritable metabolic fluxes.
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