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BReast CAncer (BRCA1 and BRCA2) is a tumor suppressor gene that plays an important role in the repair of double strand DNA break (DSB) via error-free homologous recombination (HR). BRCA1 and BRCA2 mutations induce vulnerability to PARP inhibition, known as synthetic lethality, in breast and ovarian cancers but is rare in acute myeloid leukemia (AML).
As key players in humoral immunity, B cells play diverse roles in immune responses and disease development. Although early studies focused on the hallmark features of antibody production in B cells, increasing evidence has demonstrated the effector functions of cytokine secretion and antigen presentation by B cells in the development of various human diseases.
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