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1 Introduction Chronic Inflammatory Demyelinating Polyneuropathy (CIDP) is an autoimmune neuropathy characterized by slowly progressive weakness and sensory dysfunction in the arms and legs [1]. Despite advances in the understanding of the disease's pathophysiology, CIDP remains a challenging condition to manage, with significant variability in treatment response [2].
1 Introduction Chronic inflammatory demyelinating polyneuropathy (CIDP) causes paresis and sensory dysfunction in the extremities [1]. The primary pathophysiology is demyelination of peripheral nerves [2] and typical findings on electrophysiologic examination include decreased nerve conduction velocity and conduction blocks [3-5]. Secondary axonal loss may result in reduced compound motor action potentials (CMAP) [6].
RESEARCH REPORT This article has been accepted for publication and undergone full peer review but has not been through the copyediting, typesetting, pagination and proofreading process, which may lead to differences between this version and the Version of Record. Please cite this article as doi: 10.1111/jns.12563.
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