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Abstract WHO-endorsed shortened, all-oral regimens have improved tolerability and outcomes for MDR/RR-TB treatment, but predictors of poor outcomes remain incompletely-characterized. We conducted a prospective cohort study (NCT05871489) of adults with bacteriologically-confirmed pulmonary MDR/RR-TB treated with a shortened, all-oral regimen in Lesotho.
Background The mitochondrion is the primary target of oxidative stress in response to exogenous environments. Mitochondrial DNA (mtDNA) is independent from nuclear DNA and uses separate epigenetic machinery to regulate mtDNA methylation. The mtDNA damage induced by oxidative stress can cause mitochondrial dysfunction and is implicated in human diseases; however, mtDNA methylation has been largely overlooked in environmental studies relating to human disease.
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