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Abstract Vγ9Vδ2 T cells mediate HLA-independent targeting of tumor cells, offering unique opportunities for cancer immunotherapy, yet their therapeutic efficacy remains limited. Here, using a genome-wide CRISPR screen, we identify UDP-galactose 4-epimerase (GALE) as a tumor-intrinsic checkpoint that restricts Vγ9Vδ2 T cell-mediated killing.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
