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Articles by M. Bynoe
David Martin's "malleable" reality: the no-virus movement doesn't exist but imaginary viruses do
Greetings and Best Wishes, It’s almost as if some of the Virus Pushers Against Clotshots are in a competition to see who can come up with the most ridiculous “virus” narrative.
Searching for common ground
In the mid-1960s, while studying patients with respiratory symptoms, two pairs of scientists discovered a new type of virus unlike any they had seen before in humans ( 1 , 2 ). These viruses were later given the name “coronaviruses” owing to spike-like projections on the surface that were reminiscent of the solar corona. In the following decades, other human-infecting coronaviruses were identified, along with a wide variety of coronaviruses that infect birds and mammals ( 3 ).
Antibody Responses to SARS-CoV-2: Let’s Stick to Known Knowns
Nicole Baumgarth, Janko Nikolich-Žugich, F. Eun-Hyung Lee and Deepta Bhattacharya J Immunol November 1, 2020, 205 (9) 2342-2350; DOI: https://doi.org/10.4049/jimmunol.2000839 Abstract The scale of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pandemic has thrust immunology into the public spotlight in unprecedented ways.
COVID‐19: Emergence of Infectious Diseases, Nanotechnology Aspects, Challenges, and Future Perspectives
Abstract Wuhan, a city of China, is the epicenter for the pandemic outbreak of coronavirus disease‐2019 (COVID‐19). It has become a severe public health challenge to the world and established a public health emergency of international worry. This infectious disease has pulled down the economy of almost all top developed nations. The coronaviruses (CoVs) known for various epidemics caused time to time.
Neuropilin-1 Expression on CD4 T Cells Is Atherogenic and Facilitates T Cell Migration to the Aorta in Atherosclerosis
Dalia E. Gaddis, Lindsey E. Padgett, Runpei Wu and Catherine C. Hedrick J Immunol December 15, 2019, 203 (12) 3237-3246; DOI: https://doi.org/10.4049/jimmunol.1900245 Key Points Western diet feeding increases Nrp1 on Foxp3− CD4 T cells in ApoE−/− mice. Nrp1+Foxp3− CD4 T cells are highly proliferative and atherogenic. Nrp1+Foxp3− CD4 T cells preferentially migrate to the aorta and PaLN.
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