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To view the full text, please login as a subscribed user or purchase a subscription. Click here to view the full text on ScienceDirect. Following myocardial infarction, increased protein kinase A (PKA) and Ca2+ calmodulin-dependent kinase II (CaMKII) activities hyper-activate Ryanodine Receptor 2 (RyR2), contributing to intracellular Ca2+ dysregulation and cardiac arrhythmias.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
