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Articles by M. Rozman
Charge transfer via temporary bonds in ${\mathrm{C}}_{60} + {\mathrm{C}}_{60}^{+}$ molecular collisions
We present a theoretical description of resonant charge transfer in collisions of nanoparticles, specifically for C60+C60+ collisions. We predict that transient bonds between colliding fullerenes can significantly extend the interaction time, allowing for a greater probability of charge transfer. In our model, the dumbbell-shaped (C60−C60)+ quasimolecule, that is temporarily formed during the collision, is described as a dynamic system of 120 zero-range potentials.
Erratum: Inner energy relaxation and growth of nanosize particles [Phys. Rev. A 108, 032812 (2023)]
ISSN 2469-9934 (online), 2469-9926 (print). ©2024 American Physical Society. All rights reserved. Physical Review A™ is a trademark of the American Physical Society, registered in the United States, Canada, European Union, and Japan. The APS Physics logo and Physics logo are trademarks of the American Physical Society. Information about registration may be found here.
Afección oculta de médula ósea en la enfermedad de Hodgkin: detección por resonancia magnética
IdiomacatalàDeutschEnglishespañoleuskarafrançaisgalegoitalianoportuguêsromână
Resonancia magnética de la médula ósea vertebral: análisis densitométrico
IdiomacatalàDeutschEnglishespañoleuskarafrançaisgalegoitalianoportuguêsromână
Inner energy relaxation and growth of nanosize particles
In this study, molecular-dynamics simulations were conducted to investigate the relaxation of the internal energy in nanosized particles and its impact on the nucleation of atomic clusters. Accurate binary potentials were utilized to analyze the growth and collisional relaxation of the internal energy of ArnH+ clusters in a metastable Ar gas.
Acquired erythropoietic uroporphyria secondary to myelodysplastic syndrome with chromosome 3 alterations. A case report
Congenital erythropoietic porphyria (CEP) is a rare, autosomal recessive disease caused by a deficiency of uroporphyrinogen III synthase (UROS), due to mutations in the UROS gene in chromosome 10. On occasion, patients show a mild late-onset disease, without germline UROS mutations, associated with hematologic malignancies. We report a 65-year-old patient with photosensitivity, porphyrins overexcretion and thrombocytopenia.
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