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Abstract Retinal neovascularization (RNV) is a major cause of irreversible vision loss worldwide. Although pericyte–endothelial cell cross-talk has been implicated in RNV, the exosome-mediated mechanism underlying this interaction has remained uncharacterized. Here we found that hexokinase 2 (HK2) expression was upregulated in pericytes under hypoxic conditions both in vitro and in vivo. Hypoxic pericytes released exosomes enriched in HK2, which promoted endothelial cell growth in vitro.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
