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Abstract Placental malaria is characterized by the sequestration of Plasmodium falciparum–infected red blood cells (iRBCs) within the intervillous space through VAR2CSA-mediated adhesion to chondroitin sulfate A (CSA) expressed on trophoblasts. Although cytoadherence is a key pathogenic event, its impact on extracellular ATP (eATP) signaling remains poorly understood.
Arlisson M. Rodrigues1* Mariana T. Tanno1 Mariana M. Contti1 Hong S. Nga1 Mariana F. Valiatti1 Silvana D. Costa2 Tainá V. Freitas2 Ronaldo d. Esmeraldo2 Camila M. Assunção3 Juliana B. Tassi3 Gustavo F. Ferreira3 Claudia R. Felipe4 Jose O. Medina Pestana4 Helio T. Silva4 Luis G.
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