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Study design The aim of this study was to establish the effect of the rs7665090 risk variant on (i) astrocyte function in vitro and in situ, (ii) on MS lesion pathology, and (iii) on lesion load in MS patients. iPSC-derived astrocytes, autopsied MS cases, and MS patients were all homozygous for either the risk or protective variant. We used astrocyte cultures derived from a total of 24 iPSC lines obtained from 6 MS patients and 6 healthy donors.
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