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1 Introduction Voltage-gated sodium (Nav) channel Nav1.7 is preferentially expressed in the peripheral nervous system and is essential for action potential (AP) generation in sensory neurons due to its role in the initial signal amplification, possibly serving as a threshold channel for APs.[1] Clinical and genetic evidence shows that Nav1.7 plays an important role in the perception of pain signals.[1, 2] Accordingly, Nav1.7 is considered as an analgesic target, and pharmacological inhibition...
Open AccessArticle by Lijiang Chen 1,†, Changkun Qiao 1,†, Meijing Wu 2,†, Linghan Cai 1, Cong Yin 2, Mukun Yang 2, Xiubo Sang 2 and Wenpei Bai 2,* 1 School of Electronic and Information Engineering, Beihang University, No. 37 Xueyuan Road, Haidian District, Beijing 100191, China 2 Department of Obstetrics and Gynecology, Beijing Shijitan Hospital, Capital Medical University, Beijing 100038, China * Author to whom correspondence should be addressed.
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