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Abstract Background: Metabolic dysfunction-associated fatty liver disease (MAFLD) is a growing metabolic disorder closely linked to gut micro biota dysbiosis and impaired gut–liver communication. Dietary interventions targeting the gut micro biota represent a promising strategy for MAFLD management. Fucoidan (FUC), a marine-derived bioactive polysaccharide with limited host digestibility, is primarily metabolized by intestinal microorganisms.
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