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Abstract Cholinergic modulation of brain reward circuitry appears to play a crucial role in information processing about salience as a key biological mechanism in obesity. However, changes in acetylcholine transmission leading to abnormal eating behavior have not been demonstrated in vivo in human obesity.
1. Introduction Obesity is a major public health challenge worldwide. Understanding the processes that contribute to this pandemic is of significant clinical importance as obesity is strongly associated with an increased risk of all-cause morbidity and mortality [1]. The complex physiological process of food intake is necessary for survival and is governed by the homeostatic system and the hedonic systems in the human brain [2].
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